{"id":46627,"date":"2021-07-02T13:08:25","date_gmt":"2021-07-02T07:38:25","guid":{"rendered":"https:\/\/johnsonfrancis.org\/professional\/?p=46627"},"modified":"2021-07-02T13:08:25","modified_gmt":"2021-07-02T07:38:25","slug":"cardiology-mcq","status":"publish","type":"post","link":"https:\/\/johnsonfrancis.org\/professional\/cardiology-mcq\/","title":{"rendered":"Cardiology MCQs"},"content":{"rendered":"<p><iframe loading=\"lazy\" title=\"YouTube video player\" src=\"https:\/\/www.youtube.com\/embed\/zzpUqJgTIl4\" width=\"560\" height=\"315\" frameborder=\"0\" allowfullscreen=\"allowfullscreen\"><\/iframe><\/p>\n<h2><span style=\"color: #008000;\">Cardiology MCQs<\/span><\/h2>\n<p><strong><span style=\"color: #0000ff;\">Ischemic preconditioning is mediated by:<\/span><\/strong><\/p>\n<ol>\n<li>ATP-sensitive potassium channel (K<sub>ATP<\/sub>)<\/li>\n<li>Sodium channel<\/li>\n<li>I<sub>kr<\/sub>, the rapid component of delayed rectifier potassium current<\/li>\n<li>L-type calcium channel<\/li>\n<\/ol>\n<p><span style=\"color: #008000;\"><strong>Correct answer: 1. ATP-sensitive potassium channel (K<sub>ATP<\/sub>)<\/strong><\/span><\/p>\n<p>Blockers of ATP-sensitive potassium channel (K<sub>ATP<\/sub>) like glibenclamide can prevent ischemic reconditioning. K<sub>ATP<\/sub> channel opener nicorandil can mimic ischemic preconditioning and it is known as pharmacological preconditioning. Stimulation of adenosine receptors is known to simulate ischemic preconditioning. [Tomai F, Crea F, Gaspardone A, Versaci F, De Paulis R, Penta de Peppo A, Chiariello L, Gioffr\u00e8 PA. <a href=\"https:\/\/pubmed.ncbi.nlm.nih.gov\/8044938\/\">Ischemic preconditioning during coronary angioplasty is prevented by glibenclamide, a selective ATP-sensitive K+ channel blocker<\/a>. Circulation. 1994 Aug;90(2):700-5.]<\/p>\n<p><span style=\"color: #0000ff;\"><strong>First percutaneous intervention for a regurgitant lesion in humans was for:<\/strong><\/span><\/p>\n<ol>\n<li>Aortic regurgitation<\/li>\n<li>Mitral regurgitation<\/li>\n<li>Pulmonary regurgitation<\/li>\n<li>Tricuspid regurgitation<\/li>\n<\/ol>\n<p><span style=\"color: #008000;\"><strong>Correct answer: 4. Pulmonary regurgitation<\/strong><\/span><\/p>\n<p>Bonhoeffer P et al described the development of a system for percutaneous stent implantation combined with valve replacement for right ventricle to pulmonary artery prosthetic conduit with valve dysfunction in 2000. [Bonhoeffer P, Boudjemline Y, Saliba Z, Merckx J, Aggoun Y, Bonnet D, Acar P, Le Bidois J, Sidi D, Kachaner J. <a href=\"https:\/\/pubmed.ncbi.nlm.nih.gov\/11052583\/\">Percutaneous replacement of pulmonary valve in a right-ventricle to pulmonary-artery prosthetic conduit with valve dysfunction<\/a>. Lancet. 2000 Oct 21;356(9239):1403-5.]<\/p>\n<p><span style=\"color: #0000ff;\"><strong>Which of the following is NOT a feature of pulmonary hypertension on pulmonary valve M-Mode echocardiogram:<\/strong><\/span><\/p>\n<ol>\n<li>Flat EF slope<\/li>\n<li>Shallow a wave<\/li>\n<li>Deep a wave<\/li>\n<li>Mid systolic notch<\/li>\n<\/ol>\n<p><span style=\"color: #008000;\"><strong data-rich-text-format-boundary=\"true\">Correct answer: 3. Deep a wave<\/strong><\/span><\/p>\n<p>Deep a wave is seen in pulmonary stenosis. All others are features of pulmonary hypertension on M-Mode echocardiogram.<\/p>\n<figure id=\"attachment_25534\" aria-describedby=\"caption-attachment-25534\" style=\"width: 500px\" class=\"wp-caption aligncenter\"><img loading=\"lazy\" decoding=\"async\" class=\"wp-image-25534 size-full\" src=\"https:\/\/johnsonfrancis.org\/professional\/wp-content\/uploads\/2009\/07\/MS-PAH.jpg\" sizes=\"auto, (max-width: 500px) 100vw, 500px\" srcset=\"https:\/\/johnsonfrancis.org\/professional\/wp-content\/uploads\/2009\/07\/MS-PAH.jpg 500w, https:\/\/johnsonfrancis.org\/professional\/wp-content\/uploads\/2009\/07\/MS-PAH-300x167.jpg 300w\" alt=\"Pulmonary valve M-Mode echocardiogram in mitral stenosis with pulmonary hypertension\" width=\"500\" height=\"278\" \/><figcaption id=\"caption-attachment-25534\" class=\"wp-caption-text\">Pulmonary valve M-Mode echocardiogram in pulmonary hypertension showing flat EF slope and shallow a wave<\/figcaption><\/figure>\n<p><span style=\"color: #0000ff;\"><strong>Feature of mitral stenosis on M-Mode echocardiogram:<\/strong><\/span><\/p>\n<ol>\n<li>Flat EF slope<\/li>\n<li>Absent A wave<\/li>\n<li>Paradoxical anterior movement of posterior mitral leaflet<\/li>\n<li>All of the above<\/li>\n<\/ol>\n<p><strong><span style=\"color: #008000;\">Correct answer: 4. All of the above<\/span><\/strong><\/p>\n<figure style=\"width: 500px\" class=\"wp-caption aligncenter\"><img loading=\"lazy\" decoding=\"async\" src=\"https:\/\/johnsonfrancis.org\/professional\/wp-content\/uploads\/2010\/09\/M-mode-echocardiogram-in-mitral-stenosis1.jpeg\" alt=\"M-mode echocardiogram in severe mitral stenosis with flat EF slope\" width=\"500\" height=\"395\" \/><figcaption class=\"wp-caption-text\">M-mode echocardiogram in severe mitral stenosis with flat EF slope, absent A wave and paradoxical anterior movement of posterior leaflet<\/figcaption><\/figure>\n<figure id=\"attachment_25533\" aria-describedby=\"caption-attachment-25533\" style=\"width: 500px\" class=\"wp-caption aligncenter\"><img loading=\"lazy\" decoding=\"async\" class=\"wp-image-25533 size-full\" src=\"https:\/\/johnsonfrancis.org\/professional\/wp-content\/uploads\/2009\/07\/MS-Mode.jpg\" sizes=\"auto, (max-width: 500px) 100vw, 500px\" srcset=\"https:\/\/johnsonfrancis.org\/professional\/wp-content\/uploads\/2009\/07\/MS-Mode.jpg 500w, https:\/\/johnsonfrancis.org\/professional\/wp-content\/uploads\/2009\/07\/MS-Mode-300x188.jpg 300w\" alt=\"M mode echocardiogram in mitral stenosis \" width=\"500\" height=\"314\" \/><figcaption id=\"caption-attachment-25533\" class=\"wp-caption-text\">Reduced EF slope, absent A wave and paradoxical anterior movement of posterior mitral leaflet in mitral stenosis<\/figcaption><\/figure>\n<p><span style=\"color: #0000ff;\"><strong>Which coronary vein is important in the genesis of atrial fibrillation?<\/strong><\/span><\/p>\n<ol>\n<li>Great cardiac vein<\/li>\n<li>Oblique vein of Marshall<\/li>\n<li>Middle cardiac vein<\/li>\n<li>Small cardiac vein<\/li>\n<\/ol>\n<p><span style=\"color: #008000;\"><strong>Correct answer: 2. Oblique vein of Marshall<\/strong><\/span><\/p>\n<p>Oblique vein of Marshall, also known as oblique vein of left atrium, can give rise to atrial fibrillation. Three reasons described are the myocardial extensions into the structure, node like remnants within the vein and the rich autonomic innervation surrounding it [Habib A, Lachman N, Christensen KN, Asirvatham SJ. <a href=\"https:\/\/pubmed.ncbi.nlm.nih.gov\/19861386\/\">The anatomy of the coronary sinus venous system for the cardiac electrophysiologist<\/a>. Europace. 2009 Nov;11 Suppl 5:v15-21].<\/p>\n<p><span style=\"color: #0000ff;\"><strong>Valve at the ostium of coronary sinus is known as:<\/strong><\/span><\/p>\n<ol>\n<li>Thebesian valve<\/li>\n<li>Vieussen\u2019s valve<\/li>\n<li>Eustachian valve<\/li>\n<li>None of the above.<\/li>\n<\/ol>\n<p><span style=\"color: #008000;\"><strong>Correct answer: 1. Thebesian valve<\/strong><\/span><\/p>\n<p>Valve at the ostium of the posterolateral vein at the junction of the great cardiac vein and coronary sinus is called Vieussen\u2019s valve. Eustachian valve is at the ostium of the inferior vena cava, which directs blood to the foramen ovale in fetal life.<\/p>\n<p><strong><span style=\"color: #0000ff;\">Greatest sensitivity for ST elevation in right ventricular infarction is for ST elevation in:<\/span><\/strong><\/p>\n<ol>\n<li>V1<\/li>\n<li>V2<\/li>\n<li>V3R<\/li>\n<li>V4R<\/li>\n<\/ol>\n<p><strong><span style=\"color: #008000;\">Correct answer: 4. V4R<\/span><\/strong><\/p>\n<p>ST elevation of 1 mm or more in V4R had the greatest sensitivity of 93% and predictive accuracy (93%) for right ventricular myocardial infarction [Braat SH, Brugada P, de Zwaan C, Coenegracht JM, Wellens HJ. V<a href=\"https:\/\/pubmed.ncbi.nlm.nih.gov\/6299315\/\">alue of electrocardiogram in diagnosing right ventricular involvement in patients with an acute inferior wall myocardial infarction<\/a>. Br Heart J. 1983 Apr;49(4):368-72].<\/p>\n<p><strong><span style=\"color: #0000ff;\">Which of the following is NOT a usual clinical finding in right ventricular infarction?<\/span><\/strong><\/p>\n<ol>\n<li>Elevated jugular venous pressure<\/li>\n<li>Positive Kussmaul&#8217;s sign<\/li>\n<li>Hypotension<\/li>\n<li>Basal crepitation<\/li>\n<\/ol>\n<p><strong><span style=\"color: #008000;\">Correct answer: 4. Basal crepitation<\/span><\/strong><\/p>\n<p>Lung fields are usually clear in right ventricular infarction despite elevated jugular venous pressure. This is because the pulmonary capillary pressure is usually low due to reduced right ventricular output and left ventricular preload [Berger PB, Ryan TJ. <a href=\"https:\/\/pubmed.ncbi.nlm.nih.gov\/2404629\/\">Inferior myocardial infarction. High-risk subgroups<\/a>. Circulation. 1990 Feb;81(2):401-11].<\/p>\n","protected":false},"excerpt":{"rendered":"<p>Cardiology MCQs Ischemic preconditioning is mediated by: ATP-sensitive potassium channel (KATP) Sodium channel Ikr, the rapid component of delayed rectifier potassium current L-type calcium channel Correct answer: 1. ATP-sensitive potassium channel (KATP) Blockers of ATP-sensitive potassium channel (KATP) like glibenclamide can prevent ischemic reconditioning. KATP channel opener nicorandil can mimic ischemic preconditioning and it is [&hellip;]<\/p>\n","protected":false},"author":1,"featured_media":34621,"comment_status":"closed","ping_status":"closed","sticky":false,"template":"","format":"standard","meta":{"nf_dc_page":"","footnotes":""},"categories":[3,5],"tags":[],"class_list":["post-46627","post","type-post","status-publish","format-standard","has-post-thumbnail","hentry","category-cardiology-mcq","category-dm-dnb-cardiology-entrance"],"yoast_head":"<!-- This site is optimized with the Yoast SEO plugin v27.9 - https:\/\/yoast.com\/product\/yoast-seo-wordpress\/ -->\n<title>Cardiology MCQs - All About Cardiovascular System and Disorders<\/title>\n<meta name=\"robots\" content=\"index, follow, max-snippet:-1, max-image-preview:large, max-video-preview:-1\" \/>\n<link rel=\"canonical\" href=\"https:\/\/johnsonfrancis.org\/professional\/cardiology-mcq\/\" \/>\n<meta property=\"og:locale\" content=\"en_US\" \/>\n<meta property=\"og:type\" content=\"article\" \/>\n<meta property=\"og:title\" content=\"Cardiology MCQs - All About Cardiovascular System and Disorders\" \/>\n<meta property=\"og:description\" content=\"Cardiology MCQs Ischemic preconditioning is mediated by: ATP-sensitive potassium channel (KATP) Sodium channel Ikr, the rapid component of delayed rectifier potassium current L-type calcium channel Correct answer: 1. 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