New Series of Interactive Cardiology MCQs 8
Advanced Clinical Series
Cardiology MCQs: Part 8
Challenge yourself with master-level concepts in hemodynamics, structural interventions, EP, and advanced cardiovascular imaging.
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1. During right heart catheterization, the presence of giant, prominent ‘v waves’ in the pulmonary capillary wedge pressure (PCWP) tracing is most indicative of:
2. Idiopathic Ventricular Tachycardia originating from the Right Ventricular Outflow Tract (RVOT) classically presents on a 12-lead ECG with:
3. Which landmark clinical trial demonstrated a mortality benefit for using transcatheter edge-to-edge repair (TEER, e.g., MitraClip) in highly selected patients with severe secondary (functional) mitral regurgitation and heart failure?
4. In patients with a high bleeding risk (HBR) who undergo percutaneous coronary intervention (PCI) with a modern drug-eluting stent (DES) for stable angina, current guidelines suggest Dual Antiplatelet Therapy (DAPT) can be safely shortened to:
5. On Cardiac Magnetic Resonance (CMR) imaging, the calculation of Extracellular Volume (ECV) via T1 mapping is most drastically elevated in which of the following cardiomyopathies?
6. The underlying pathophysiological mechanism of pulsus paradoxus in cardiac tamponade is primarily driven by:
7. In patients with Wolff-Parkinson-White (WPW) syndrome, what differentiates Antidromic AVRT from Orthodromic AVRT on a 12-lead ECG?
8. What is the primary FDA-approved indication for percutaneous Left Atrial Appendage (LAA) closure devices (e.g., Watchman)?
9. Which class of medications recently became the first to definitively show a reduction in cardiovascular death and heart failure hospitalizations in patients with Heart Failure with PRESERVED Ejection Fraction (HFpEF)?
10. In Hypertrophic Obstructive Cardiomyopathy (HOCM), the dynamic left ventricular outflow tract (LVOT) obstruction is primarily caused by asymmetric septal hypertrophy and what other essential echocardiographic finding?
11. Ebstein anomaly is a congenital heart defect most frequently associated with which distinct electrocardiographic finding?
12. The Brockenbrough-Braunwald-Morrow sign, observed during cardiac catheterization, describes:
13. Commotio cordis causes sudden cardiac death in young athletes following blunt chest trauma primarily by triggering Ventricular Fibrillation via which electrophysiological mechanism?
14. Adenosine slows AV nodal conduction primarily by agonizing which specific receptor?
15. In which of the following scenarios is Surgical Aortic Valve Replacement (SAVR) generally preferred over Transcatheter Aortic Valve Replacement (TAVR) according to current guidelines?
16. During a nuclear myocardial perfusion stress test (SPECT), the finding of Transient Ischemic Dilation (TID) of the left ventricle post-stress is highly suggestive of:
17. According to ACC/AHA guidelines, which profile represents a Class I indication for Cardiac Resynchronization Therapy (CRT/Biventricular Pacing)?
18. When calculating cardiac output in the cath lab using the estimated Fick method, what is the most common source of calculation error?
19. Arrhythmogenic Right Ventricular Cardiomyopathy (ARVC) is fundamentally a genetic disease of which myocardial structure?
20. Which of the following is the most lethal, albeit less common, adverse side effect of long-term Amiodarone therapy?
21. In classic Rheumatic Mitral Stenosis, the morphological hallmark that leads to the narrowing of the valve orifice is primarily:
22. A 45-year-old symptomatic patient presents to the ER with chest pain and receives a Coronary Artery Calcium (CAC) score of 0. Why can obstructive coronary artery disease NOT be completely ruled out?
23. For optimal hemodynamic support, an Intra-Aortic Balloon Pump (IABP) should be timed to:
24. In a patient with a known genetic predisposition for Brugada Syndrome, which of the following common systemic stressors is a well-known trigger that can acutely unmask the Type 1 Brugada ECG pattern and induce fatal arrhythmias?
25. Cilostazol is frequently used to treat the symptoms of intermittent claudication in peripheral artery disease. What is its exact mechanism of action?
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About The Author
Johnson Francis
Former Professor of Cardiology, Calicut Govt. Medical Kozhikode, Kerala, India. Editor-in-Chief, BMH Medical Journal